PIPELINE
Our researchers, assisted by world class facilities and ground-breaking scientific platforms, are continually evolving and improving our science, their knowledge and capabilities. This has fueled a strong pipeline spanning several therapy areas that we are confident will deliver highly differentiated solutions.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Bimekizumab is an investigational humanized monoclonal IgG antibody that selectively inhibits both IL-17A and IL-17F, two key cytokines driving inflammatory processes.
Zilucoplan is an investigational macrocyclic peptide inhibitor of complement component 5 (C5). The U.S. Food and Drug Administration (FDA) has granted Orphan Drug Designation to zilucoplan for the treatment of myasthenia gravis.
Zilucoplan is an investigational macrocyclic peptide inhibitor of complement component 5 (C5). The U.S. Food and Drug Administration (FDA) has granted Orphan Drug Designation to zilucoplan for the treatment of myasthenia gravis.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Rozanolixizumab is an investigational humanized monoclonal antibody that specifically binds to human neonatal Fc receptor (FcRn). It has been designed to block the interaction of FcRn and IgG, inhibiting IgG recycling and inducing the removal of pathogenic IgG autoantibodies.
Dapirolizumab pegol is an investigational humanised monovalent pegylated Fab antibody fragment against the CD40 ligand (CD40L). Through interactions with its receptor, CD40, CD40L plays an important role in regulating interactions between T cells and other immune cells and thus affects several important functional events thought to be involved in autoimmune disease.
Dapirolizumab pegol is being co-developed with Biogen.
Dapirolizumab pegol is an investigational humanised monovalent pegylated Fab antibody fragment against the CD40 ligand (CD40L). Through interactions with its receptor, CD40, CD40L plays an important role in regulating interactions between T cells and other immune cells and thus affects several important functional events thought to be involved in autoimmune disease.
Dapirolizumab pegol is being co-developed with Biogen.
Staccato® Alprazolam is an orally-inhaled investigational therapy designed to be used as a single-use epileptic seizure rescue therapy that combines the Staccato® delivery technology with alprazolam, a benzodiazepine.
Staccato® Alprazolam is an orally-inhaled investigational therapy designed to be used as a single-use epileptic seizure rescue therapy that combines the Staccato® delivery technology with alprazolam, a benzodiazepine.
Bepranemab is an investigational recombinant, humanised, full length IgG4 monoclonal anti-tau antibody with specificity for human tau protein.
Bepranemab is being co-developed with Roche/Genentech
Bepranemab is an investigational recombinant, humanised, full length IgG4 monoclonal anti-tau antibody with specificity for human tau protein.
Bepranemab is being co-developed with Roche/Genentech
UCB0599 is an investigational small molecule that prevents the pathological misfolding and accumulation of alpha-synuclein, a protein which plays a role in Parkinson’s disease (PD) pathology. Inhibition of alpha-synuclein misfolding has the potential to slow down the progression of PD.
UCB0599 belongs to a series of molecules discovered by Neuropore, which were in-licensed by UCB in 2014.
UCB0599 is being co-developed with Novartis.
UCB0599 is an investigational small molecule that prevents the pathological misfolding and accumulation of alpha-synuclein, a protein which plays a role in Parkinson’s disease (PD) pathology. Inhibition of alpha-synuclein misfolding has the potential to slow down the progression of PD.
UCB0599 belongs to a series of molecules discovered by Neuropore, which were in-licensed by UCB in 2014.
UCB0599 is being co-developed with Novartis.